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Chronic allograft nephropathy – a clinical syndrome: early detection and the potential role of proliferation signal inhibitors

Identifieur interne : 007F11 ( Main/Exploration ); précédent : 007F10; suivant : 007F12

Chronic allograft nephropathy – a clinical syndrome: early detection and the potential role of proliferation signal inhibitors

Auteurs : Josep M. Campistol [Espagne] ; Ioannis N. Boletis [Grèce] ; Jacques Dantal [France] ; Johan W. De Fijter [Pays-Bas] ; Alexandre Hertig [France] ; Hans H. Neumayer [Allemagne] ; Ole Yen [Norvège] ; Julio Pascual [Espagne] ; Erich Pohanka [Autriche] ; Juan C. Ruiz [Espagne] ; Maria P. Scolari [Italie] ; Sergio Stefoni [Italie] ; Daniel Ser N [Espagne] ; Vito Sparacino [Italie] ; Wolfgang Arns [Allemagne] ; Jeremy R. Chapman [Australie]

Source :

RBID : ISTEX:17B049A6CAC81CAD63C7568C00D6DF472B53400F

Descripteurs français

English descriptors

Abstract

Abstract:  Chronic allograft nephropathy (CAN) leads to the majority of late graft loss following renal transplantation. Detection of CAN is often too late to permit early intervention and successful management. Most current strategies for managing CAN rely on minimizing or eliminating calcineurin inhibitors (CNIs) once CAN has become established. The proliferation signal inhibitors everolimus and sirolimus have potent immunosuppressive and antiproliferative actions, with the potential to alter the natural history of CAN by reducing CNI exposure whilst avoiding acute rejection. Whilst data will be forthcoming from a number of clinical trials investigating this potential, we discuss early detection of CAN and the rationale for a role for this class of agent.

Url:
DOI: 10.1111/j.1399-0012.2009.01057.x


Affiliations:


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Le document en format XML

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<term>Acute rejection</term>
<term>Allograft</term>
<term>Allograft dysfunction</term>
<term>Allograft nephropathy</term>
<term>Antibacterial agent</term>
<term>Biopsy</term>
<term>Calcineurin</term>
<term>Calcineurin inhibitor</term>
<term>Calcineurin inhibitors</term>
<term>Campistol</term>
<term>Cell proliferation</term>
<term>Chronic</term>
<term>Chronic allograft dysfunction</term>
<term>Chronic allograft nephropathy</term>
<term>Chronic rejection</term>
<term>Chronic renal failure</term>
<term>Clinical syndrome</term>
<term>Cnis</term>
<term>Creatinine</term>
<term>Creatinine clearance</term>
<term>Cyclosporine</term>
<term>Diagnosis</term>
<term>Dial</term>
<term>Dose reduction</term>
<term>Dysfunction</term>
<term>Early detection</term>
<term>Early intervention</term>
<term>Early intervention strategies</term>
<term>Early stage</term>
<term>Everolimus</term>
<term>Glomerular filtration</term>
<term>Graft</term>
<term>Graft loss</term>
<term>Graft survival</term>
<term>Histological</term>
<term>Homograft</term>
<term>Homotransplantation</term>
<term>Immunosuppression</term>
<term>Immunosuppressive</term>
<term>Immunosuppressive agent</term>
<term>Immunosuppressive regimen</term>
<term>Immunosuppressive therapies</term>
<term>Inhibitor</term>
<term>Kidney</term>
<term>Kidney disease</term>
<term>Kidney transplantation</term>
<term>Late graft loss</term>
<term>Ltration</term>
<term>Ltration rate</term>
<term>Mammalian target of rapamycin</term>
<term>Mofetil</term>
<term>Mycophenolate</term>
<term>Mycophenolate mofetil</term>
<term>Nankivell</term>
<term>Negative slope</term>
<term>Nephrol</term>
<term>Nephrol dial transplant</term>
<term>Nephropathy</term>
<term>Potential role</term>
<term>Proliferation</term>
<term>Proteinuria</term>
<term>Protocol biopsies</term>
<term>Rate</term>
<term>Regimen</term>
<term>Rejection</term>
<term>Renal</term>
<term>Renal failure</term>
<term>Renal function</term>
<term>Renal transplant patients</term>
<term>Renal transplant recipients</term>
<term>Renal transplantation</term>
<term>Risk factors</term>
<term>Serum creatinine</term>
<term>Signal transduction</term>
<term>Sirolimus</term>
<term>Slope analysis</term>
<term>Syndrome</term>
<term>Tacrolimus</term>
<term>Transplant</term>
<term>Transplant proc</term>
<term>Transplant recipients</term>
<term>Transplantation</term>
<term>Treatment</term>
</keywords>
<keywords scheme="Pascal" xml:lang="fr">
<term>Antibactérien</term>
<term>Chronique</term>
<term>Diagnostic</term>
<term>Evérolimus</term>
<term>Filtration glomérulaire</term>
<term>Fonction rénale</term>
<term>Homogreffe</term>
<term>Homotransplantation</term>
<term>Immunodépresseur</term>
<term>Insuffisance rénale</term>
<term>Insuffisance rénale chronique</term>
<term>Multiplication cellulaire</term>
<term>Néphropathie</term>
<term>Pathologie du rein</term>
<term>Prolifération</term>
<term>Protéine mTOR</term>
<term>Rein</term>
<term>Rejet</term>
<term>Stade précoce</term>
<term>Syndrome</term>
<term>Taux</term>
<term>Traitement</term>
<term>Transduction signal</term>
<term>Transplantation rénale</term>
</keywords>
<keywords scheme="Teeft" xml:lang="en">
<term>Acute rejection</term>
<term>Allograft</term>
<term>Allograft dysfunction</term>
<term>Allograft nephropathy</term>
<term>Biopsy</term>
<term>Calcineurin</term>
<term>Calcineurin inhibitor</term>
<term>Calcineurin inhibitors</term>
<term>Campistol</term>
<term>Chronic allograft dysfunction</term>
<term>Chronic allograft nephropathy</term>
<term>Chronic rejection</term>
<term>Clinical syndrome</term>
<term>Cnis</term>
<term>Creatinine</term>
<term>Creatinine clearance</term>
<term>Cyclosporine</term>
<term>Dial</term>
<term>Dose reduction</term>
<term>Dysfunction</term>
<term>Early detection</term>
<term>Early intervention</term>
<term>Early intervention strategies</term>
<term>Everolimus</term>
<term>Graft</term>
<term>Graft loss</term>
<term>Graft survival</term>
<term>Histological</term>
<term>Immunosuppression</term>
<term>Immunosuppressive</term>
<term>Immunosuppressive regimen</term>
<term>Immunosuppressive therapies</term>
<term>Inhibitor</term>
<term>Kidney transplantation</term>
<term>Late graft loss</term>
<term>Ltration</term>
<term>Ltration rate</term>
<term>Mofetil</term>
<term>Mycophenolate</term>
<term>Mycophenolate mofetil</term>
<term>Nankivell</term>
<term>Negative slope</term>
<term>Nephrol</term>
<term>Nephrol dial transplant</term>
<term>Nephropathy</term>
<term>Potential role</term>
<term>Proteinuria</term>
<term>Protocol biopsies</term>
<term>Regimen</term>
<term>Renal</term>
<term>Renal function</term>
<term>Renal transplant patients</term>
<term>Renal transplant recipients</term>
<term>Renal transplantation</term>
<term>Risk factors</term>
<term>Serum creatinine</term>
<term>Sirolimus</term>
<term>Slope analysis</term>
<term>Tacrolimus</term>
<term>Transplant</term>
<term>Transplant proc</term>
<term>Transplant recipients</term>
<term>Transplantation</term>
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<front>
<div type="abstract">Abstract:  Chronic allograft nephropathy (CAN) leads to the majority of late graft loss following renal transplantation. Detection of CAN is often too late to permit early intervention and successful management. Most current strategies for managing CAN rely on minimizing or eliminating calcineurin inhibitors (CNIs) once CAN has become established. The proliferation signal inhibitors everolimus and sirolimus have potent immunosuppressive and antiproliferative actions, with the potential to alter the natural history of CAN by reducing CNI exposure whilst avoiding acute rejection. Whilst data will be forthcoming from a number of clinical trials investigating this potential, we discuss early detection of CAN and the rationale for a role for this class of agent.</div>
</front>
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<li>Australie</li>
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<li>Nouvelle-Galles du Sud</li>
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<li>Oslo</li>
<li>Paris</li>
<li>Sydney</li>
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<name sortKey="Chapman, Jeremy R" sort="Chapman, Jeremy R" uniqKey="Chapman J" first="Jeremy R." last="Chapman">Jeremy R. Chapman</name>
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